Overview

HOW TO LOSE WEIGHT AND INCREASE YOUR ENERGY NATURALLY

THE AVATRIM PROGRAM WILL HELP YOU:

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FIXING THE PROBLEMS WITH YOUR METABOLISM THAT CAUSE WEIGHT GAIN

There are 3 main parts to the Avatrim program the Formula, the Food Plan, and the Activity Plan. Each of these three parts acts individually to positively influence the user's metabolism, in various ways. Taken together, the overall effect of all three major program parts on metabolism is cumulative and very powerful and the reason why Avatrim is called the only correct way to lose weight.

LOSE FAT PERMANENTLY

It's a fact - 95% of the people who go on conventional diets gain all the weight back and sometimes they end up fatter than when they started. Now you can learn how to be in the successful 5% group that keeps it off forever.

LOSE BODY FAT WITHOUT WRECKING YOUR METABOLISM

If you've ever started a diet, then smashed into the dreaded "plateau," it's probably because you cannibalized your own muscle and slowed down your metabolism. Not only will this system teach you the only way to prevent your metabolism from crashing, you will also learn more than a dozen ways to fire up your "metabolic engine" and accelerate your body's natural rate of calorie-burning.

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Studies

CLINICAL STUDIES

In a study reported on in the American Journal of Clinical Nutrition, it was found that green tea extract resulted in a significant increase in energy expenditure, plus also had a significant effect on fat oxidation. While some of the effects were originally theorized to be due to the caffeine content of green tea, the researchers discovered that the tea actually has properties that go beyond those that would be explained by the caffeine.

Today, scientific research in both Asia and the west is providing hard evidence for the health benefits long associated with drinking green tea. For example, in 1994 the Journal of the National Cancer Institute published the results of an epidemiological study indicating that drinking green tea reduced the risk of esophageal cancer in Chinese men and women by nearly sixty percent. University of Purdue researchers recently concluded that a compound in green tea inhibits the growth of cancer cells. There is also research indicating that drinking green tea lowers total cholesterol levels, as well as improving the ratio of good (HDL) cholesterol to bad (LDL) cholesterol.

In a study reported on in the American Journal of Clinical Nutrition, it was found that green tea extract resulted in a significant increase in energy expenditure (a measure of metabolism), plus also had a significant effect on fat oxidation. While some of the effects were originally theorized to be due to the caffeine content of green tea, the researchers discovered that the tea actually has properties that go beyond those that would be explained by the caffeine.

The same amount of caffeine as was in the green tea, administered alone, failed to change energy expenditure in other studies. This led reseachers to believe that there is some interaction going on with the active ingredients of green tea that promotes increased metabolism and fat oxidation.

The researchers indicated that their findings have substantial implications for weight control. A 4% overall increase in 24-hour energy expenditure was attributed to the green tea extract, however, the research found that the extra expenditure took place during the daytime. This led them to conclude that, since thermogenesis (the body's own rate of burning calories) contributes 8-10% of daily energy expenditure in a typical cubject, that this 4% overall increase in energy expenditure due to the green tea actually translated to a 35-43% increase in daytime thermogenesis.

Of critical importance to thyroid patients is the fact that none of the research subjects reported any side effects, and no significant differences in heart rates were noticed. In this respect, green tea extract is different from some of the prescription drugs for obesity, and herbal products like ephedra, which can raise heart rates and blood pressure, and are not recommended for many individuals, in particular, those with thyroid disease who may be particularly sensitive to stimulants.

SOURCE: Nagao, T. American Journal of Clinical Nutrition, January 2005; vol 81: 122-129.

References

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2. Lee WJ, Song KH, Koh EH, Won JC, Kim HS, Park HS, Kim MS, Kim SW, Lee KU, Park JY. Alpha-lipoic acid increases insulin sensitivity by activating AMPK in skeletal muscle. Biochem Biophys Res Commun. 2005;332(3):885-887

3. Shixian Q, VanCrey B, Shi J, Kakuda Y, Jiang Y. Green tea extract thermogenesis-induced weight loss by epigallocatechin gallate inhibition of catechol-O-methyltransferase. J Med Food. 2006;9(4):451-8

4. Malini T, Arunakaran J, Aruldhas MM, Govindarajulu P. Effects of piperine on the lipid composition and enzymes of the pyruvate-malate cycle in the testis of the rat in vivo. Biochem Mol Biol Int. 1999;47(3):537-45

5. Lee SW,Rho MC, Park HR ,et al .Inhibition of diacylglycerol acyltransferase by alkamides isolated from the fruits of Piper longum and Piper nigrum. J Agric Food Chem. 2006;54(26):9759-63

6. Bitar MS, Wahid S, Pilcher CW,Al-Saleh E, Al-Mulla F. Alpha-lipoic acid mitigates insulin resistance in Goto-Kakizaki rats. Horm Metab Res. 2004;36(8):542-9..

7. Reddi A.; DeAngelis B.; Frank O.; Lasker N.; Bakura H. Biotin supplementation improves glucose and insulin tolerances in genetically diabetic KK mice. LIFE SCI. 1988, 42(13):1323-1330

8. Zhang H.; Osada K.; Maebashi M.,Ito M., Komai M., Furukawa Yand H. ZhangA high biotin diet improves the impaired glucose tolerance of long-term spontaneously hyperglycemic rats with non-insulin-dependent diabetes mellitus. Journal of Nutritional Science and Vitaminology (Japan).1996;42(6):517-526

9. O'Meara S, Riemsma R, Shirran L, Mather L, ter Riet G. A rapid and systematic review of the clinical effectiveness and cost-effectiveness of orlistat in the management of obesity. Health Technol Assess. 2001; 5(18):1-81.

10. Sayama K et al. Effects of green tea on growth, food utilization and lipid metabolism in mice. In Vivo 2000; 14(4):481-4.

11. Wang X et al. "Green tea epigallocatechin gallate: a natural inhibitor of fatty-acid synthase." Biochem Biophys Res Commun2001;288: 1200-1208

12. Cooper R, Moore DJ and Morre DM. medicinal beneifits of green tea:Part I. review of non-cancer benefits. J Altern Complement Med. 2005;11(3):521-52822.

13. Murase T et al. "Beneficial effects of tea catechins on diet-induced obesity: stimulation of lipid catabolism in the liver. Int J Obes Relat Metab Disord. 26, 11:1459-64, 2002.

14. Koutsikos D.; Fourtounas C.; Kapetanaki A., et al .Oral glucose tolerance test after high-dose i.v. biotin administration in normoglucemic hemodialysis patients. Renal Failure. 1996;18(1):131-137

15. Kim MS, Park JY, Namkoong C et al. Anti-obesity effects of alpha-lipoic acid mediated by suppressionof hypothalamic AMP-activated protein kinase. Nat Med. 2004;10(7):727-33

16. Jacob S, Rues P, Hermann R. Oral administration of RAC-alpha-lipoic acid modulates insulin sensitivity in patients with type-2 diabetes mellitus: a placebo-controlled pilot trial. Free Rad Biol Med. 1999;27(3-4):309-14

17. Moines H, Trios O, Park YC, Chow KJ, Packer L R-alpha-Lipoic Acid Action on Cell Redox Status, the Insulin Receptor, and Glucose Uptake in 3T3-L1 Adipocytes. Arch Biochem Biophys. 2002;15:397(2): 384-91.

18. Korotchkina LG, Sidhu S and Patel MS. R-lipoic acid inhibits mammalian pyruvate dehydrogenase kinase. Free Radical Res. 2004;38:1083-1092

19. WHO. Available on. http://www.who.int/mediacentre/factsheets/fs311/en/index.html Accessed on 10/02/07

20. CDC. Available at. http://www.cdc.gov/nccdphp/dnpa/obesity/faq.htm#doing Accessed on 10/02/07

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22. National Health and Nutrition Examination Survey (NHANES). Available at. http://win.niddk.nih.gov/publications/PDFs/stat904z.pdf Accessed on 10/02/2007.

Ingredients

INGREDIENTS

Green tea and Camellia Sinensis Leaf